Search results for "ACID UPTAKE"

showing 2 items of 2 documents

Liver and pancreatic fat content and metabolism in healthy monozygotic twins with discordant physical activity

2011

Background & Aims: Ectopic fat in muscle and liver is linked to obesity and type 2 diabetes. Recently, pancreatic lipid accumulation has also been associated with beta-cell dysfunction and reduced insulin production, leading to the development of type 2 diabetes. Physical exercise training has been shown to attenuate beta-cell dysfunction in patients, but little is known about its effects on pancreatic and hepatic fat accumulation. In this study, we validated in-vivo proton magnetic resonance spectroscopy ((1)H MRS) in pancreatic fat measurement with biochemical measurements in a pig model. Thereafter, the effects of increased physical activity on the amounts of pancreatic and liver fat…

MaleSwinemedicine.medical_treatmentSus scrofaAdipose tissueMonozygotic twinACID UPTAKEType 2 diabetesFatty Acids NonesterifiedFat Measurement0302 clinical medicineFatty AcidsHEPATIC INSULIN-RESISTANCEMagnetic Resonance Imagingmedicine.anatomical_structureADIPOSE-TISSUEAdipose TissueLiverModels AnimalSwine Miniature030211 gastroenterology & hepatologyPancreasMonozygotic twinsAdultmedicine.medical_specialty030209 endocrinology & metabolismDEPENDENT DIABETES-MELLITUSMotor ActivityBiologyta3111HISPANIC ADOLESCENTSYoung Adult03 medical and health sciencesInsulin resistanceBETA-CELL FUNCTIONInternal medicineMagnetic resonance spectroscopymedicineMAGNETIC-RESONANCE SPECTROSCOPYAnimalsHumansADIPONECTIN CONCENTRATIONSPancreasPLASMA ADIPONECTINHepatologyPhysical activityInsulinTRIGLYCERIDE CONTENTTwins MonozygoticLipid Metabolismmedicine.diseaseObesityEndocrinologyInsulin ResistanceJournal of Hepatology
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VEGF-B-induced vascular growth leads to metabolic reprogramming and ischemia resistance in the heart

2014

Abstract Angiogenic growth factors have recently been linked to tissue metabolism. We have used genetic gain‐ and loss‐of function models to elucidate the effects and mechanisms of action of vascular endothelial growth factor‐B (VEGF‐B) in the heart. A cardiomyocyte‐specific VEGF‐B transgene induced an expanded coronary arterial tree and reprogramming of cardiomyocyte metabolism. This was associated with protection against myocardial infarction and preservation of mitochondrial complex I function upon ischemia‐reperfusion. VEGF‐B increased VEGF signals via VEGF receptor‐2 to activate Erk1/2, which resulted in vascular growth. Akt and mTORC1 pathways were upregulated and AMPK downregulated, …

VEGF‐Bmedicine.medical_specialtyMedicine (General)AngiogenesiseducationMOUSE HEARTIschemiaVEGF-B610 Medicine & healthmTORC1ischemiaBiologyQH426-470CONTRIBUTESchemistry.chemical_compoundangiogenesisR5-920CARDIAC-FUNCTIONInternal medicinemedicineGeneticsFAILUREta318Myocardial infarctionFATTY-ACID UPTAKEREPERFUSION INJURY610 Medicine & healthProtein kinase BMYOCARDIAL HYPERTROPHYAMPKta3121medicine.diseaseCell biologyARTERIOGENESISVascular endothelial growth factorMICEEndocrinologychemistry3121 General medicine internal medicine and other clinical medicineendothelial cellMolecular Medicine3111 BiomedicineReperfusion injurymetabolism
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